2014 October

USA Health: Carnivorousness and Vegetarianism

Carnivorousness and Vegetarianism vegeterian

Meat has introduced such poisons in your organism that today it is a unity of impurities. If you had a normal sense of smell, you would stay far from each other – the smell of those, who eat meat, is so bad. When somebody tells me that he is ailing, the first condition for improvement of his health is to change the food he eats. “What shall we eat then?” The answer is – fruits.

What will one gain if he eats? Will he become stronger. However, if he eats vegetable food, he will become cleaner. Carnivorous animals are stronger, more cruel, more predatory. If you are a wolf, you will eat meat. If you are a sheep, you will eat grass. The desires of the wolf are desires of the Black Lodge, and the desires of the sheep are desires of the White Lodge.

Carnivorousness is the disobedience of God’s law.

In order to understand which food is healthier, let us do the following experiment: we will put somebody on a diet – three months we will give him to eat only pork and three times a day he will drink half a liter of wine. We will put another on a fruit diet. In the course of two months he will eat exclusively fruits and drink clean hot water. We will watch both of them during the experiment and will see what their relationships with their relatives and people round them will be.

You may eat meat and cheese, but they cannot create in you a clean body, a noble mind.

One, who cannot avoid meat, cannot get rid of evil. Blood is purified through food. A full vegetarian may be the one, whose ancestors, the previous four or five generations, were vegetarians.

Pigs are the most unclean animals, and so is their meat. One thinks that together with his gut disposal all impurities are removed. Wheat is the purest food.

People die, because carnivorousness exists.

I deny meat, because I know that all crimes throughout the world are due only to it. One, who eats meat, will suffer for sure.

The extreme killing of mammals creates an anomaly in Nature. Most diseases are due to that killing, which stops their evolution, and all powers that shall create their welfare remain unused, thus forming a chaotic state, which is the reason for the various diseases. Do you know what happens after the animal blood flows out promiscuously? From the evaporation – various serums and favorable conditions for harmful germs form and from there evils in the organic world come.

No one, who eats pork or goat, may become great. John the Baptist ate honey and locusts. He was very energetic. Honey softened his character. Locusts imparted gaiety and good mood GlobalCanadianPharmacy.

You have studied fruit-eating with angels, and you have studied carnivorousness with the fallen spirits. Carnivorousness came into the world when it was broken the connection of the human soul with Cod, with Love.

By admin on October 30, 2014 | Men's Health

The pathophysiology of chronic asthma is complex

The pathophysiology of chronic asthma is complex, in that it involves smooth-muscle dysfunction, acute and chronic inflammation, and structural changes within the airway, collectively termed airway remodeling. It has generally been assumed that airway inflammation is responsible for the various manifestations of asthma including shortness of breath, wheezing, bronchial hyperresponsiveness, smooth-muscle dysfunction and, ultimately, structural changes. However, inflammation is not the sole mediator of this disease. As effective as inhaled glucocorticoid therapy is, it alone is often insufficient to adequately control asthma in patients with moderate-to-severe persistent asthma. 4

In addition, multiple studies have shown that doubling the dose of inhaled glucocorticoid in patients inadequately controlled on inhaled glucocorticoid therapy Generic Viagra pharmacy alone fails to provide significant improvement in efficacy, while increasing the potential for systemic and adverse effects. This unresponsiveness calls into question the paradigm that airway inflammation is solely responsible for the manifestations of asthma and that glucocorticoid-independent mechanisms need to be considered. Whether this reflects our limited understanding of the pathogenesis of airway remodeling and/or a lack of effective therapies to target glucocorticoid-insensitive mechanisms remain an open issue.

The literature describing the pathology of severe, steroid-dependent childhood asthma is virtually nonexistent. This case series is among the first to combine historical and clinical features, pulmonary physiology, and endobronchial biopsy results in a group of children with severe steroid-dependent asthma. All of the subjects studied had a long, if not lifelong Cialis in Canada history of severe, persistent, high-risk asthma refractory to aggressive management that included long-term oral and high-dose inhaled glucocorticoid therapy.

A majority of these patients had been intubated at least once due to severe asthma exacerbations. In every case, the endobronchial biopsies revealed significant changes to the structure of the airways, with little to no airway inflammation present. These findings suggest that noninflam-matory-mediated mechanisms may contribute to severe asthma in children.

By admin on October 17, 2014 | Asthma, Critical Care

In Vivo Animal Study and Cisplatin Level in Blood and Tissue

No animal died of airway complications during the period of study. Intermittent stridor was noted at times of agitation in two rabbits; bronchoscopy of these animals showed a moderate amount of airway secretion. The activity and appetite of all animals were normal. Obvious weight gain was noted during the period of study. Bronchoscopic pictures revealed excellent lumen patency, with only minimal airway secretions in most of the animals. We did not observe stent fragmentation in any of the animals. Furthermore, no evidence of fac-ture at the surfaces or at the welded spots was noted for the biodegradable stents.

The in vivo release characteristics of the biodegradable drug-eluting stents Viagra Online. The cisplatin levels in the stented trachea were measurable from the first week and stably maintained at high levels until the end of the observation period (5 weeks). Similar trends were observed in the trachea segment near the stent and over the right upper lung parenchyma. In contrast, the serum levels of cisplatin were low throughout the experiment.

The trachea with stent in place. Grossly, the stented area showed mild edematous change with mucosa congestion. Microscopically, there were marked mononuclear cell infiltrates of various proportions of lymphocytes, plasma cells, and eosinophils in the submucosa of the stented area 1 week after implantation. The cilia of the epithelium were preserved. Similar findings were noted at weeks 3 and 5.

SEMSs have been of great value in palliation of malignant airway obstruction but are also notorious for various troublesome complications. This is partly due to the embedded nature of the stent but is also related to the lack of effective locoregional therapy for endobronchial lesions. Given the promising results from several EITC studies, we believe that a biodegradable stent that exerts strong stenting force and sustainable drug-eluting characteristics would be an ideal combination. In the current study, we successfully demonstrated that the biodegradable PCL stents have mechanical strength comparable to the strength of SEMSs, with minimal tissue reaction. Furthermore, for the first time, to our knowledge, through the combined use of PLGA materials and the spray coating technique, a controlled amount of cisplatin could be released locally over a few weeks with minimal systemic concentration.

By admin on | Critical Care

Radiographic Fibrosis

It is unknown if the radiographic fibrosis score predicts mortality in persistent hypersensitivity pneumonitis (HP) and if survival is similar to that observed in idiopathic pulmonary fibrosis (IPF) when adjusting for the extent of radiographic fibrosis.

We reviewed records from 177 patients with HP and 224 patients with IPF whose diagnoses were established by multidisciplinary consensus. Two thoracic radiologists scored high-resolution CT (HRCT) scan lung images. Independent predictors of transplant-free survival were determined using a Cox proportional hazards analysis. Kaplan-Meier survival curves were constructed, stratified by disease Viagra 100mg pills  as well as fibrosis score.

HRCT scan fibrosis score and radiographic reticulation independently predicted time to death or lung transplantation. Clinical predictors included a history of cigarette smoking, auscultatory crackles on lung examination, baseline FVC, and FEV1/FVC ratio. The majority of HP deaths occurred in patients with both radiographic reticulation and auscultatory crackles on examination, compared with patients with only one of these manifestations (P < .0001). Patients with IPF had worse survival than those with HP at any given degree of radiographic fibrosis (hazard ratio 2.31; P < .01).

Survival in patients with HP was superior to that of those with IPF with similar degrees of radiographic fibrosis. The combination of auscultatory crackles and radiographic reticulation identified patients with HP who had a particularly poor outcome.

Hypersensitivity pneumonitis (HP) is a diffuse parenchymal lung disease characterized by an immunologic reaction to an inhaled organic antigen. Patients with HP, especially those with acute manifestations, generally have a favorable outcome, particularly when the causative antigen is identified and removed. However, patients may alternatively develop a chronic form of disease that can be characterized by the presence of fibrosis on histopathologic or radiographic evaluation. Data from the National Center for Health Statistics show that age-adjusted mortality rates from HP have increased significantly (P < .0001) from 0.09 to 0.29 per million between 1980 and 2002 in US adults. Both radiographic and pathologic fibrosis are associated with an increase in all-cause mortality in HP, but it is unclear if these are independent risk factors when adjusting for other clinical and physiologic predictors.

By admin on October 9, 2014 | Asthma

Potential risk factors for diagnosis of ABPA

Potential risk factors for diagnosis of ABPA examined in this analysis included sex; age at enrollment; percent of predicted FVC and FEV1 using the equations of Knudson et al; the presence of wheeze on physical examination at the enrollment visit; a diagnosis of asthma within the preceding 6 months; and positive culture for any Pseudomonas at enrollment or within the preceding year.

For calculation of ABPA prevalence rates and prevalence rates of risk factors, the denominators included all patients > 5 years of age enrolled in ESCF with valid data for sex, age, and medical conditions. Prevalence rates were calculated by dividing the number of patients in whom ABPA was diagnosed by the total number of CF patients at risk. The importance Viagra Australia online of possible risk factors and strength of association was estimated using multiple logistic regression. Age, FVC, and FEV1 were entered into the model as categorical variables rather than continuous variables because exploratory analysis indicated that ABPA risk did not increase linearly in association with these variables.


Between December 1993 and May 1996, 14,210 patients aged > 5 years enrolled in ESCF. The frequency distributions for patients’ demographic and disease characteristics are shown in Table 2. Slightly more male than female patients were in this analysis. The mean (SD) age was 17.1 (9.6) years, with a median of 14.7 years. Pulmonary function test data are summarized, for the patients who had reported test values, in Table 2. Pulmonary function measurements were not reported for 6% of the patients. In those with data, the mean (SD) FVC was 84%(25) of predicted value (median, 86%), and the mean (SD) FEVi was 71%(28) of predicted value (median, 73%). Wheezing on physical examination at enrollment was noted in 11% of subjects; asthma was diagnosed in 19% in the 6 months preceding enrollment; and a positive culture for Pseudomonas spp was reported in 62% in the year preceding enrollment.

ABPA was diagnosed in 281 patients (2%) in the 6 months preceding enrollment. In the children < 5 years of age participating in ESCF (n = 3,796), only one case of ABPA was reported. This age group was therefore not included in the analysis. Table 3 shows the regional differences in prevalence of ABPA. The overall reported prevalence rate of ABPA in North America was 2.0%. Regional rates ranged from the low of 0.9% in the Southwest to the high of 4.0% in the western region of the United States. Canada and the other U.S. regions had intermediate prevalence rates.

By admin on October 2, 2014 | Critical Care, Sexual Health